These findings were consistent with the posterior reversible encephalopathy syndrome. We report our conventional magnetic resonance and diffusion-weighted imaging findings and briefly discuss the pathophysiology of the syndrome.”
“Objective-To determine whether administration of 2 doses of a multivalent, modified-live virus vaccine prior to breeding of heifers would provide protection against abortion and fetal infection following exposure of pregnant heifers to cattle persistently infected LY294002 (PI) with bovine viral diarrhea virus (BVDV) and cattle with acute bovine herpesvirus 1 (BHV1) infection.
Design-Randomized controlled clinical trial.
Animals-33
crossbred beef heifers, 3 steers, 6 bulls, and 25 calves.
Procedures-20 of 22 vaccinated and 10 of 11 unvaccinated heifers became pregnant and were
commingled with 3 steers PI with BVDV type 1a, 1b, or 2 for 56 days beginning 102 days after the second vaccination (administered 30 days after the first vaccination). Eighty days following HDAC inhibitor removal of BVDV-PI steers, heifers were commingled with 3 bulls with acute BHV1 infection for 14 days.
Results-After BVDV exposure, 1 fetus (not evaluated) was aborted by a vaccinated heifer; BVDV was detected in 0 of 19 calves from vaccinated heifers and in all 4 fetuses (aborted after BHV1 exposure) and 6 calves from unvaccinated heifers. Bovine herpesvirus 1 was not detected in any 3 Methyladenine fetus or calf and associated fetal membranes in either treatment group. Vaccinated heifers had longer gestation periods and calves with greater birth weights, weaning weights, average daily gains, and market value at weaning, compared with those for calves born to unvaccinated heifers.
Conclusions
and Clinical Relevance-Prebreeding administration of a modified-live virus vaccine to heifers resulted in fewer abortions and BVDV-PI offspring and improved growth and increased market value of weaned calves. (J Am Vet Med Assoc 2012;241:484-495)”
“The defect structure of Ge(111) epilayers grown by molecular beam epitaxy on cubic Pr2O3(111)/Si(111) support systems was investigated by means of transmission electron microscopy and laboratory-based x-ray diffraction techniques. Three main types of defects were identified, namely, rotation twins, microtwins, and stacking faults, and studied as a function of Ge film thickness and after annealing at 825 degrees C in ultrahigh vacuum. Rotation twins were found to be localized at the Ge(111)/cubic Pr2O311 (1) over bar interface and their amount could be lowered by the thermal treatment. Microtwins across 11 (1) over bar were detected only in closed Ge films, after Ge island coalescence.